We will start with a neuropathology table proposing that co-pathology is the norm in dementia, and most patients have several components: tau, amyloid, tdp43, alpha-synuclein.
CHAIRPERSON: Alberto Rábano . BTCIEN-Fundación CIEN.
Gabor Kovacs . Laboratory Medicine & Pathobiology. University of Toronto. (On-line)
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TBD
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In the following table, we will address how genetic variability is associated with the risk of AD and with most of the frequent AD co-pathologies (alpha-synuclein, TDP43, vascular…) and can potentially be applied to diagnostic and/or risk classification. We will also explore brain resilience from a genetic perspective.
CHAIRPERSON: Agustín Ruiz
Alfredo Ramírez , Cluster of Excellence for Aging Research. University of Cologne.
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Henne Holstege , Alzheimer Center Amsterdam. VUmc. Amsterdam UMC
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We will deepen into neuroimaging techniques for detecting different dementia-related patterns reflecting the disease components.
CHAIRPERSON: Michel Grothe
David Wolk , Laboratory for Cognition and Neural Stimulation. University of Pennsylvania
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Jesús Silva. , Fundación CIEN
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Laura Jonkman, Amsterdam Neuroscience - Brain Imaging (VUmc). Amsterdam UMC
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Discussing the detection of neuropathological components in the periphery using biochemical markers.
CHAIRPERSON: Pascual Sánchez Juan Fundación CIEN
Gemma Salvadó , Clinical Memory Research. Lund University
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Anja Schneider, Translational Dementia Research. German Center for Neurodegenerative Diseases-DZNE. University of Bonn.
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